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Nevertheless, most first-line drugs for the treatment of those disorders target the serotonin and/or dopamine systems either directly (e.g., in depression) or indirectly via modulation of GABA-A receptors

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Different mechanisms are pro-posed to explain curcumin's effect on glucose homeostasis, which are mainly based on the activation of the gamma receptor, which is activated by the peroxisome proliferator (PPAR-y)

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