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Structure and Function of the 26S Proteasome
Individual differences in susceptibility to idiosyncratic drug-induced liver injury may reflect genetic differences in either (a) alternate metabolic pathways by which a drug is converted to different (potentially hepatotoxic) metabolites, (b) the ability of the individual to detoxify the toxic intermediates, (c) an underlying immunologic or allergic reaction, or (d) an individual's tolerance or ability to adapt to hepatocellular injury (mechanisms unknown) with resolution of injury despite continued medication administration.20, 135 Oral medications with substantial hepatic metabolism are more likely to be associated with adverse hepatic events in humans, presumably because of hepatic generation of reactive toxic metabolites
10.1034/j.1600-079x.2000.280407.x 77 MartnM.MacasM.LenJ.EscamesG.KhaldyH.Acua-CastroviejoD
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