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285 Based on the above phenomena, researchers have developed therapeutic ideas to target Gln metabolism in tumor cells, increase the concentration of Gln in TME, and promote Gln metabolism in tumor-infiltrating cells

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[76] found tuberculosis (TB) treatment via blocking Mycobacterium tuberculosis (Mtb) protein tyrosine phosphatase A -host protein arginine methyltransferase 6 interface to target GPX4-dependent ferroptosis, eventually inducing ferroptosis to promote Mtb pathogenicity and dissemination

FOXO4-DRI shows potential as a treatment targeting the senescence and apoptosis resistance, and holds promise as an approach to prevent the aggressiveness and relapse of keloids