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In conclusion, the accumulation of specific uremic toxins in CKDsuch as indoxyl sulfate, phosphate, vanadate, and acroleinpromotes eryptosis through distinct but convergent mechanisms involving Ca 2+ dysregulation, oxidative stress, energy depletion, and kinase activation

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In this case glutathione is not a specific coenzyme but rather a natural antioxidant that can be synthesized in the cell from readily available precursors