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treatment of blood cells with lead resulted in ROS-independent eryptosis without any increase in ROS, but with an increase in Ca 2+ level and caspase-3 activity

This dual antioxidant mechanism explains GHK-Cu's protective effects in inflammatory conditions and its ability to enhance cellular survival under stress conditions

Experimental evidence from murine CCl 4 -induced liver fibrosis models suggests that neutrophils may also participate in fibrosis resolution by secreting MMP8 and MMP9 (100) ( Figure 4E )