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many negative cardiovascular outcomes have been linked to exposure levels below current safe standards ( 19 , 14 , 15 )

(121) published in 2024 revealed a novel mechanism by which AhR, as a uremic toxin receptor, promotes renal senescence and fibrosis: IS upregulates and activates AhR in renal tubular epithelial cells, and AhR can interact with peroxisome proliferator-activated receptor gamma coactivator 1-alpha (PGC1), promoting its ubiquitin-mediated degradation through its E3 ubiquitin ligase activity, thereby inhibiting mitochondrial biogenesis

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Effect of nitric oxide production on the redox modulatory site of the NMDA receptor-channel complex