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Open Biochem J 2014;8:83-8

Modulation of glutamate agonist-induced influx of calcium into neurons by -l-glutamyl and -l-aspartyl dipeptides

In support of this increased NK cytotoxic potential, the expression of the NK cytotoxic effector-associated gene perforin, as determined by flow-cytometric analysis or quantitative real-time PCR analysis in CD56 + NK cells, was also significantly increased in the above IGF-1-induced CD34 + -derived NK cells compared with the cells without IGF-1 (Fig