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Description

Neuron 81 , 1234 (2014)

This is an educational research reference

This leads to the release and cytoplasmic translocation of p53, which can subsequently activate transcription-independent pro-apoptotic signaling (Clara et al., 2024

This observation has been reported in earlier studies in obese and hyperinsulinemic subjects, and in patients with Cushings syndrome, in whom hepatic sensitivity to GH is increased when compared to lean subjects (95, 103)