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CD28-dependent Rac1 activation is the molecular target of azathioprine in primary human CD4+ T lymphocytes

Moreover, overexpression of cGAS in RA-FLSs enhanced both the proliferation of these cells and the expression of pro-inflammatory factors ( Sting1 +/ mice, which have a reduced expression of phosphorylated TBK1, showed a decreased severity of arthritis and improved histological changes compared to control mice ( Figure 2 )

It was also observed to protect neurons under stress, reduce reactive oxygen species (ROS), and inhibit apoptosis