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In addition to developing therapeutic agents that target the reprogramming of ammonium metabolism in prostate cancer, it is essential to identify reliable indicators for evaluating treatment efficacy, such as sensitivity-related gene signatures and metabolomic profiling (277)

Glutamine antagonism, aiming to disrupt tumor growth by targeting critical metabolic pathways, is challenged by the adaptive nature of cancer cells and the complex metabolic microenvironment, potentially compromising its therapeutic efficacy

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