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Latest advances in dual inhibitors of acetylcholinesterase and monoamine oxidase B against Alzheimers disease

(2018) showed that acute ischemic myelin injury is initiated by vesicular release from axons, which results in cytotoxic overactivation of myelinic NMDA receptors comprising GluN2C/D subunits, which are primarily incorporated into extra-synaptic NMDA receptors (Brickley et al., We should also mention the cysteine-glutamate exchanger (xCT), which promotes glutathione synthesis by taking up cysteine and releasing glutamate, and which could possibly add to glutamate excitotoxicity evoked by ischemia and, consequently, to the overactivation of extrasynaptic NMDA receptors and neuronal death

On the other hand, chronic kidney disease causes fast excretion of cobalamin through urine, resulting in low levels

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