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In vivo , administration of shikonin in severely immunodeficient murine EMs models significantly reduces monocyte infiltration into the peritoneal cavity

1c, d) 42,58 , a rather unspecific GSH-dependent monothiol mechanism might be better suited for the reduction of a variety of physiological non-glutathione disulfide substrates to maintain metabolites and proteins in a reduced state (Fig

In addition, the result of immunofluorescence staining for wound healing assay after treatment with HGF showed that the expression of p-Met of BxPC-3 cells that migrated to the gap front was higher (Supplementary Fig

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