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However, portal insulin delivery is also an essential co-player in the regulation of the GH/IGF-I axis by affecting and regulating hepatic GH receptor synthesis, and subsequently altering hepatic GH sensitivity and IGF-I generation

This is followed by goblet cell depletion, erosion, ulceration, and infiltration of neutrophils into the lamina propria and submucosa (81), triggering the innate immune response (82, 83)