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Thus, the uptake of AA by SVCT2, with the consequent phosphorylation and activation of ERK1/2, could be responsible for the expression of genes for neurogenesis, differentiation, and synaptic maturation observed in studies of DNA microarrays in stem cells, ventral precursors stimulated by AA (Yu et al., 2004) and in cortical neurons that overexpress SVCT2 ( 7

The study was conducted with 41 male rats that received contusion injuries or sham procedures

10 Reduced glutathione is associated with higher oxidative stress, 11 which contributes to damage 12 in the trabecular meshwork cells and retinal ganglion cells, worsening glaucoma and accelerating optic nerve damage