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Copper complexes, such as the Hinokitiol copper complex, inhibit the deubiquitinase activity of certain proteasomes, contributing to ATF4-associated ER stress and subsequently to paraptosis [147]

Diseases associated with iron overload Iron overload may arise from genetic predispositions, such as hereditary hemochromatosis [25] or ferroportin disease [26], as well as from acquired factors, including repeated red blood cell (RBC) transfusions due to anemia, increased intestinal iron absorption and ineffective erythropoiesis [27]

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High K + levels around astrocytic end-feet increase their local K + uptake and induce K + efflux in distant cell processes [67]