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Reactive astrocytes also release neurotoxic saturated lipids through the APOJ and the C3 complement component, thus affecting neuronal network activity via C3aR signaling (Lian et al., 2015

Oxidative stress on mitochondrial antioxidant defense system in the aging process: Role of dl--lipoic acid and L-carnitine

Published mechanisms include upregulation of growth hormone receptor expression, promotion of angiogenesis via VEGF pathway activation, modulation of the nitric oxide system, and interaction with the dopaminergic and serotonergic systems in neurological models