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TB-500 via actin-mediated endothelial migration), and TB-500's signature property, systemic cell recruitment, is exactly what BPC-157 does not do
Thus, resolving a hemostatic defect, the hallmark of which is a markedly prolonged bleeding time, BPC 157 reduced the bleeding times in the tail amputation model, heparin and partly warfarin rats, but unlike in normal rats, did not reduce bleeding time in heparin and warfarin rats when previously L-arginine or L-NAME was given
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