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In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]

Migration index and migration rate were also calculated using the following 2 equations respectively: [25] MNs fabrication MNs were prepared using micro-molding technology with a polymeric matrix composed of 20% w/w PVA, 25% w/w PVP, and 0.5% w/w propolis, based on a previously reported method with minor modifications [26]

Hong M, Rong J, Tao X, Xu Y