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This is also why a secretagogue approach is framed as "axis-friendly": because endogenous somatostatin is still in the loop, negative feedback is preserved, which in principle restrains the supraphysiologic IGF-1 spikes seen with exogenous GH

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In summary, this section not only untangled the diverse molecular mechanisms by which small molecules target the regulation of MMPs to inhibit GBM invasion (involving key signaling pathways, metabolism, non-coding RNAs and ion channels), but also highlighted the considerable clinical translational potential of certain compounds [such as NP16 and novel Pt(IV) complexes] with excellent BBB penetration and in vitro/in vivo activity in overcoming GBM invasiveness (Table SIII)