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Our work stems from our previous observations that mice with whole-body disruption of GSH production (Gclm-KO) develop fewer, smaller tumors

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87 reported that targeted overexpression of eNOS in apolipoprotein E knockout animals accelerated rather than decreased atherosclerotic lesion formation

For instance, individuals with cognitive impairment due to unrelated conditions, such as neurosyphilis, HIV-associated encephalopathy, or neurocysticercosis, would not plausibly respond to anti-amyloid therapies