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In line with this, overexpression of wildtype METLL16 enhanced the expression of CBS with 3UTR-1 mutation but not with 3UTR-2 or 3UTR-3 mutation, further confirming the importance of these two sites in controlling CBS expression (Fig

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This is compelling and appealing to most scientists because its aligned with whats been thought of in the last several decades, said Martin Picard, a mitochondria biologist at Columbia University who was not involved in the study