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Critically, this redox adaptation is a key mechanism of resistance to radiotherapy and many chemotherapeutic agents, such as cisplatin and doxorubicin, which depend on generating oxidative damage to exert their cytotoxic effects [33]

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What exists instead is mechanistic plausibility: tirzepatide drives fat loss at rates faster than most patients have experienced before, and glutathione is the endogenous compound responsible for managing oxidative debris at the cellular level

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