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Moreover, besides binding of minor fraction of Cu(II) ions, His can also act as a catalyst enhancing the rate of the copper transfer between CuHSA and other proteins and cellular destinations, which might be the most important physiological role of His in copper metabolism

Instead, GHK-Cu appeared to act as a broad-spectrum homeostatic regulator, capable of resetting gene expression patterns in cultured cells towards those characteristic of a younger, more regenerative state

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