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10.1111/1541-4337.12518 [DOI] [PubMed] [Google Scholar] Coppi, F

A compensatory anti-inflammatory mechanism then typically follows (59), causing an imbalance in immune responses (58, 59) combined with hypothesised autoimmunity (52, 85, 182)

BPC-157 has been reported in animal models to promote angiogenesis, up-regulate growth-factor and receptor expression in tendon fibroblasts, and exert cytoprotective effects on the gut and other tissues with proposed involvement of the nitric-oxide system and the VEGFR2AkteNOS pathway [2] [3]