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Anticonvulsant activity of BmK AS, a sodium channel site 4-specific modulator

The decreased levels of DMG could be ascribed to the decreased levels of betaine because DMG is produced from betaine (Supplementary Fig

[10] It is worth being exact about what that source says, because the naive reading goes too far in the other direction: the recommendation is to adjust the parenteral copper dose according to serum concentrations and the clinical picture, explicitly not to stop it, since discontinuing copper in cholestatic patients risks producing copper deficiency instead

The possible mechanism that regulates the muscle phenotype is mitochondrial biogenesis and its role in tissue development (De Luca et al., 2015)