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Wide use of the rat model for studies of hippocampus-dependent learning (Blackshaw et al., 2003) and drug-induced malaise at high systemic doses of some NOS inhibitors (Prendergast et al., 1997) may also have contributed to variability in results

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259 Furthermore, endotoxin dose-dependent switch of NAD + biosynthesis pathways from NAMPT-dependent salvage to IDO1-dependent de novo biosynthesis maintains the nuclear NAD + pool, which promotes SIRT1-directed epigenetic regulation of immune tolerance