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A complicating factor in the concept of homosynaptic plasticity is the nature of input-specificity. Conventionally, plasticity is called input-specific if no changes are observed in an independent test input, not stimulated during the induction, i.e., no heterosynaptic changes

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In fibroblast-centric models, readouts consistent with collagen and matrix component regulation have been reported, supporting the use of GHK-Cu as a laboratory tool in matrix biology research