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It is not surprising that both sulfation- and glutathione-dependent pathways are aberrant in the same population because they are metabolically connected [41,72,73]

The precise mechanism of how chronic ethanol exposure is modulating AD pathology is yet to be fully uncovered

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Although several researches have evaluated the characteristics of in vivo metabolic kinetics, material clearance, tissue accumulation, and potential toxicity of materials used to induce UV, the boundary between the impact of different doses of nanoparticles on tumor growth through UV generation and the possibility of tissue damage also needs further demonstration [236, 238, 240]