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This results in an overproduction of pro-inflammatory cytokines such as interleukin 6 (IL-6), tumour necrosis factor , adhesion molecules, prostaglandins, and mitogen-activated protein kinase signalling cascades

[1] In experimental models, the peptide appears to influence several pathways involved in healing: angiogenesis, fibroblast migration, collagen organization, nitric oxide signaling, extracellular matrix formation, and reducing inflammatory cytokines

The first requirement is to generate ribose 5-phosphate for the synthesis of nucleotides, and the other is to provide reducing power in the form of NADPH

Expression of metE is repressed by a cobalamin riboswitch, while MetH requires the cobalamin cofactor