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It seems likely that sesamol protective influence on gastric mucosal injury may be at least partially through an inhibition of mucosal reactive nitrogen species and subsequent lipid peroxidation

Short-lived enzymesubstrate interactions without stable complex formation are also in accordance with infinite k cat values and an inefficient competitive inhibition of ScGrx7

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When it interacts with Nitric Oxide, it coats the endothelial tissue with a protective lining that offers a wound-healing effect