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Matrix metalloproteinase-9 activation in hyperhomocysteinemic rats is triggered by the opening of the mitochondrial permeability transition pore and antagonism of the N-methyl-D-aspartate receptor-1 (NMDA-R1), resulting in myocyte dysfunction due to elevated calcium overload and oxidative stress [40]

Further studies showed that combining MT-2 with naltrexone, a commonly used drug for treating alcohol abuse, can increase the observed activity of naltrexone in reducing binge-like alcohol drinking

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