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This review aims to explore the mechanisms by which periodontitis promotes depression through the regulation of oral and gut microbiota, as well as potential therapeutic strategies
Recently, it was shown that recombinant human GSTs, in particular GSTP1, significantly contributed to the inactivation of AQ-derived QIs by catalyzing GSH-conjugation (Zhang et al., 2017a) ( Figure 1 )
Although pharmacological compounds aimed at inducing ferroptosis, such as erastin and RSL3, or inhibiting it, such as ferrostatin-1 (fer-1), have demonstrated potential in preclinical studies, their application in clinical settings faces challenges, including off-target effects and limited tissue specificity [2,3,4,5,6]
In an attempt to overcome this, as number of methods have looked at ways in which oxygen levels can be replenished, and what makes the use of biomaterials in such applications particularly exciting is the ability to manipulate the biomaterials to gain different results
