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The enzyme is now known to be identical to GSTO1-1 (Board and Anders, 2007)

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Using this approach, the model estimates were updated to better describe busulfan exposure in both neonates and children undergoing HCT, resulting in a significantly improved fit (cohort C) as compared to the first-generation population PK model supported by the software (cohort B)

This blend may be incorporated into research involving: These applications reflect structured laboratory environments focused on endocrine system signaling and peptide interaction