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Subsequent studies have extended the functional spectrum of histone lactylation beyond immune regulation to developmental and differentiation contexts, which has been observed in embryonic stem-cell differentiation, endometrial reconstruction, and osteogenic development [86,87,88]

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However, it can cause kidney damage, leading to impaired renal function, increased BUN and SCr levels, reduced glomerular filtration, and histopathological changes like mesangial cell hypercellularity, glomerular atrophy, tubular necrosis, degeneration, and vacuolization 7,8