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922 In AKT-hyperactivated cancer cells, AKT directly phosphorylates the Ser343 site of TRPML1, inhibits the K552 ubiquitination degradation of TRPML1, and promotes the binding of TRPML1 to ARL8B, leading to lysosomal exocytosis

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The melatonin administration before or alongside CP treatment preserved the ovarian reserve by maintaining the number of primordial, primary, and growing follicles, reducing follicular atresia, and activating the Hippo signal pathway (Xu et al