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If upregulation of citrate synthase occurred due to excess PPA in vivo and then PPA was removed in vitro , the high activity of citrate synthase in vitro would overproduce citrate and enhance the proximal portion of the TCAC, which preferentially produces NADH, the electron carrier metabolized by complex I

[1] The molecular signaling that links receptor engagement to sustained IGF-1 elevation is explored in our reference on how signaling networks drive tesamorelin-induced IGF-1 elevation

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mRNA for 4F2hc was expressed in both cells, although more strongly in the resistant cells